Electron Sensitive Membranes and their Role in the
Formation of Cancer
As a general rule, every cell in your body is operating under a
system of “feedback control.” A control system of this type is
frequently found among living cells.
In trying to explain the complexity of living cells, perhaps the
best approach is to simply say that the thermostat in your home is
much like the feedback mechanisms in cells.
Without actually analyzing the components in detail, it is sufficent
to know that parts of each cell normally function together, in
reacting to whatever environment the cell is normally found in.
Secondly, DNA is the genetic apparatus which controls the functions
and activities of the living cell. That is, the same DNA is active
in each columnar epithelial cell which lines the intestine of any
given individual, as shown above.
And as a general rule, it is the “regular orientation” or “the
standardized activity” of the DNA in each of those cells which
enables them to work in a regular, stablized manner.
WHY ALL THE CONFUSION ABOUT CANCER?
It has been difficult for researchers to understand how a given
tissue has the ability to order, and/or properly restore its own
cells.
It is only now being recognized and published, by Neu Facts, Inc.,
that normal cells must communicate with each other by way of
Electron Sensitive Membrane Proteins.
These “ESM Proteins,” in turn, are linked into the feed-back control
systems, at various levels of activity, in each cell. Since each
cell as shown above, is operating under the same active DNA , then
it is also true that the same “ESM Proteins” must exist along the
sides of every one of those cells.
Hence, each cell in any given tissue is normally linked, via its
“ESM Proteins,” to the common information content of every other
cell.
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When you put cells into a regular laboratory culture however, these
cell-to-cell communications are evidentily no longer active or
essential. The entire system fails when each cell begins to
function on an independent basis.
THE DANGER OF A “HIGH-FAT DIET.
It is easy to see how fats, and especially poly-unsaturated oils
play an important part in all this. They are difficult to digest.
The fats also coat the membrane surface of the intestinal cells.
And if ANY laboratory animal is put on a high fat diet, mouse or
rat, it will spontaneously develop a cancer and it is usually a
cancer of the colon!
1/24/1990
The results of a high fat diet links perfectly to the issue of the
“ESM Proteins. The key factor is the difficulty which cells
experience in degrading fats.
As the levels of undigestible fat climb, it is obvious that they
physically limit regular “ESM Protein” activity.
Hence, the functionally connected feedback system of cellular
regulation is increasingly potentiated to fail. It poses a serious
problem.
Abnormal growth occurs if a cell (or cells) CAN NO LONGER SEND OUT
an electronic signal TO CONFIRM ITS PRESENCE among the other cells
of a given tissue. When this happens in colon cancers, most
Doctor’s know what usually takes place.
A small group of unnecessary cells starts growing first. These form
what is known as a polyp.
Assume, for the moment, that the cells normal ESM Protein
performance has been altered by the fats and that the cells normal
communications system has failed in some of the cells on both sides
of the long arrow in the drawing above.
The other cells start dividing! They are designed to reproduce but
now they are attempting to replace WHAT IS MISTAKENLY PERCEIVED TO
BE DEAD OR otherwise MISSING, neighbor cells. A glob of fat has won
a very tiny battle, but the real war doesn’t stop there.
Those cells will continue to grow until the neck of that same area
of cells is totally closed off. And as the enclosed cells continue
to grow, they will cause further problems.
If the patient is fortunate, by seeing a Doctor when the first signs
of bleeding occur, the polyps can be surgically removed. Then a
small sample of those cells, from the polyps, are sent to a
laboratory.
Once examined under a microscope, if there are no abnormal cells
growing among those cells, the growth will be declared “benign,” or
harmless.
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The Doctor’s say, “We caught it just in time.” What this means is:
the genetic apparatus of those cells (the DNA) seems to be in good
working order, and a great many cells were in one place where only a
few should have been.
What Makes Cancer Cells Different?
The abnormal operation of the cell’s DNA, is the critical factor and
once again that issue is linked to high dietary fats.
In January, 1971, M. J. Hill and associates of St. Mary’s Hospital
Medical School in London, England, discovered that certain gut
bacteria might play an important part in the emergence of colon
cancers, based on studies on cancer prone populations.
In August of that same year the same researchers reported that one
of the same bacteria that they had suspected to be somehow able to
initiate colon cancers, was also active in breast cancers.
That bacteria, reported to be a “normal component of the human
intestinal flora” was lecithinase-negative clostridia and they BREAK
DOWN FATS.
In the process, they also produce two very powerful “look alike”
female hormones. Furthermore, it is known that the hormone levels
in man drop, due to extreme stress, or when an individual passes the
normal reproductive stages. Evidently, that’s when these bacteria
really bloom.
Researchers have found that excessive amounts of hormones (estrone
and estradiol), are carried by post-menopausal women with breast
cancer.
In addition, other tissues have been found to “behave like target
organs” for these estrogens, such as the uterus, the vagina, and the
pituitary. Cancers emerge there and the same bacteria is found
there in limited numbers.
Many other reports in the medical literature note that a hidden
relationship exists between clostridium, cancer, and the hormones
which clostridium produce.
– How Does It All Fit Together? –
The initial cancer cell emerges when the key features of regular
FEEDBACK CONTROL are LOST, MASKED or DROPPED.
It may be due to radiation, a cancer causing chemical, or the
transfer of foreign DNA from a bacteria. The cell’s own DNA becomes
altered, or infected with the bacteria’s foreign DNA.
The bacteria are smaller than a dot on this screen or printed on
this page, but their DNA causes a significant change in the ESM
Protein of the affected cell.
Once the DNA is transferred, normal cell-to-cell communications fail
and the reordered DNA enables the cancer cell to live and operate on
the bacteria’s ESM Proteins.
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The cell’s normal ESM Proteins are lost. And the body’s immune
system did not detect the ESM Proteins of the bacteria, so it will
not detect the cancer cells either!
It is also important to note these things:
1. Researchers have had such a problem with cancers because
NONE of the cancer cell features have ever fallen under the
ordinary rules of bacteriology and disease.
2. The millions of cells, which make up each cancer, have each
unfolded WITH THE SAME PROPERTIES of the first, initial
cancer cell.
3. The altered DNA reorganizes membrane features which
obviously lends themselves to a flexible and unstabilized
range of activities, relative to a tissue’s ability to order
and/or properly restore it’s own numbers.
4. The bacteria involved in each cancer can be easily
identified because they normally exist among the cancer
cells which some of their own DNA, in part, initially
produced, and
5. these bacteria also produce hormone sensitive cancers, such
as breast cancers, via “hormone carriers” which transport or
move the offending DNA through the host’s blood stream to
the hormone-sensitive cells.
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Vangard Note….
Most researchers are aware of how oil effects electricity. It
acts as an insulator due to its exceedingly high dielectric
value.
This coating of oil acts as an insulator which in turn PREVENTS
the SENSING of cells BY OTHER CELLS. Replicate then occurs
WITHOUT THE GUIDANCE of the controlling chemical and IONIC
(read electrical) interchanges.
A most interesting discovery and hypothesis which might help to
explain some of the seemingly miraculous and spontaneous
“cures” of cancer. Remissions have occurred after such simple
actions as altering the environment or diet.
If this ESM Protein hypothesis is true and the agglomeration of
fat on the walls of the cells is of sufficient quantity as
brought about through a fatty diet, then it follows that the
cells could lose touch with the master controller of that organ
or system.
If true, the concept leads to new possibilities for the
treatment of cancer as well as a complete and thorough
re-examination of old and unorthodox techniques in light of the
ESM Protein theory.
We don’t have an address for this group but the file was
downloaded from the Double Helix BBS at 212-865-7043.
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We would appreciate your thoughts on this matter.
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The group appears to be Neu Facts, Inc. for which we have no
address. They included the following request if you find the file
of interest.
Note: please help us get this news out. Make copies of this for your
friends, doctors, and patients. This work was done on an
independent basis – without grants or financal istance in any
form. Thank you for your help.